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Please use this identifier to cite or link to this item: https://accedacris.ulpgc.es/handle/10553/76355
DC FieldValueLanguage
dc.contributor.authorAlmeida González, Deliaen_US
dc.contributor.authorBrito Díaz, Buenaventuraen_US
dc.contributor.authorRodríguez Pérez, María del Cristoen_US
dc.contributor.authorGonzález Hernández, Anaen_US
dc.contributor.authorDíaz Chico, B. Nicolásen_US
dc.contributor.authorCabrera de León, Antonioen_US
dc.date.accessioned2020-12-04T13:44:56Z-
dc.date.available2020-12-04T13:44:56Z-
dc.date.issued2010en_US
dc.identifier.issn0165-2478en_US
dc.identifier.otherWoS-
dc.identifier.urihttps://accedacris.ulpgc.es/handle/10553/76355-
dc.description.abstractAutoimmune diseases occur more in women than in men, and this may be attributable to the role of estrogens. Androgens promote autoimmune diseases with a profile of type 1 cytokines, such as rheumatoid arthritis, whereas estrogens promote autoimmune diseases with a type 2 cytokine profile, like systemic lupus erythematosus. Both androgens and estrogens regulate the Th1/Th2 balance. Type 1 autoimmune diseases are improved when decrease type 1 cytokines (i.e. during fasting), or when there is a rise in type 2 cytokines (increased estrogens, as in pregnancy). Type 2 autoimmune diseases improve when type 2 cytokines are diminished (decreased estrogen, as in post-partum period) or when type 1 response is stimulated.en_US
dc.languageengen_US
dc.relation.ispartofImmunology Lettersen_US
dc.sourceImmunology Letters [ISSN 0165-2478], v. 133 (1), p. 6-13, (Septiembre 2010)en_US
dc.subject320502 Endocrinologíaen_US
dc.subject.otherAutoimmune Diseasesen_US
dc.subject.otherSex Hormonesen_US
dc.subject.otherTh1/Th2 Balanceen_US
dc.subject.otherCytokinesen_US
dc.subject.otherSystemic Lupus Erythematosusen_US
dc.titleSex hormones and autoimmunityen_US
dc.typeinfo:eu-repo/semantics/reviewen_US
dc.typeReviewen_US
dc.identifier.doi10.1016/j.imlet.2010.07.001-
dc.identifier.scopus77955851131-
dc.identifier.isi000281880600002-
dc.contributor.authorscopusid8279180900-
dc.contributor.authorscopusid12788580900-
dc.contributor.authorscopusid8702688000-
dc.contributor.authorscopusid56881665800-
dc.contributor.authorscopusid57213076699-
dc.contributor.authorscopusid7006344449-
dc.identifier.eissn1879-0542-
dc.description.lastpage13en_US
dc.identifier.issue1-
dc.description.firstpage6en_US
dc.relation.volume133en_US
dc.investigacionCiencias de la Saluden_US
dc.type2Reseñaen_US
dc.contributor.daisngid29918401-
dc.contributor.daisngid1519391-
dc.contributor.daisngid27812973-
dc.contributor.daisngid2696001-
dc.contributor.daisngid6183033-
dc.contributor.daisngid14738520-
dc.description.numberofpages8en_US
dc.utils.revisionSíen_US
dc.contributor.wosstandardWOS:Gonzalez, DA-
dc.contributor.wosstandardWOS:Diaz, BB-
dc.contributor.wosstandardWOS:Perez, MDR-
dc.contributor.wosstandardWOS:Hernandez, AG-
dc.contributor.wosstandardWOS:Chico, BND-
dc.contributor.wosstandardWOS:de Leon, AC-
dc.date.coverdateSeptiembre 2010en_US
dc.identifier.ulpgcSíen_US
dc.description.jcr2,511
dc.description.jcrqQ3
dc.description.scieSCIE
item.fulltextSin texto completo-
item.grantfulltextnone-
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