1. accedaCRIS
  2. Publicaciones
  3. Reseña
Please use this identifier to cite or link to this item: https://accedacris.ulpgc.es/jspui/handle/10553/76355
DC FieldValueLanguage
dc.contributor.authorAlmeida González, Deliaen_US
dc.contributor.authorBrito Díaz, Buenaventuraen_US
dc.contributor.authorRodríguez Pérez, María del Cristoen_US
dc.contributor.authorGonzález Hernández, Anaen_US
dc.contributor.authorDíaz Chico, B. Nicolásen_US
dc.contributor.authorCabrera de León, Antonioen_US
dc.date.accessioned2020-12-04T13:44:56Z-
dc.date.available2020-12-04T13:44:56Z-
dc.date.issued2010en_US
dc.identifier.issn0165-2478en_US
dc.identifier.otherWoS-
dc.identifier.urihttps://accedacris.ulpgc.es/handle/10553/76355-
dc.description.abstractAutoimmune diseases occur more in women than in men, and this may be attributable to the role of estrogens. Androgens promote autoimmune diseases with a profile of type 1 cytokines, such as rheumatoid arthritis, whereas estrogens promote autoimmune diseases with a type 2 cytokine profile, like systemic lupus erythematosus. Both androgens and estrogens regulate the Th1/Th2 balance. Type 1 autoimmune diseases are improved when decrease type 1 cytokines (i.e. during fasting), or when there is a rise in type 2 cytokines (increased estrogens, as in pregnancy). Type 2 autoimmune diseases improve when type 2 cytokines are diminished (decreased estrogen, as in post-partum period) or when type 1 response is stimulated.en_US
dc.languageengen_US
dc.relation.ispartofImmunology Lettersen_US
dc.sourceImmunology Letters [ISSN 0165-2478], v. 133 (1), p. 6-13, (Septiembre 2010)en_US
dc.subject320502 Endocrinologíaen_US
dc.subject.otherAutoimmune Diseasesen_US
dc.subject.otherSex Hormonesen_US
dc.subject.otherTh1/Th2 Balanceen_US
dc.subject.otherCytokinesen_US
dc.subject.otherSystemic Lupus Erythematosusen_US
dc.titleSex hormones and autoimmunityen_US
dc.typeinfo:eu-repo/semantics/reviewen_US
dc.typeReviewen_US
dc.identifier.doi10.1016/j.imlet.2010.07.001-
dc.identifier.scopus77955851131-
dc.identifier.isi000281880600002-
dc.contributor.authorscopusid8279180900-
dc.contributor.authorscopusid12788580900-
dc.contributor.authorscopusid8702688000-
dc.contributor.authorscopusid56881665800-
dc.contributor.authorscopusid57213076699-
dc.contributor.authorscopusid7006344449-
dc.identifier.eissn1879-0542-
dc.description.lastpage13en_US
dc.identifier.issue1-
dc.description.firstpage6en_US
dc.relation.volume133en_US
dc.investigacionCiencias de la Saluden_US
dc.type2Reseñaen_US
dc.contributor.daisngid29918401-
dc.contributor.daisngid1519391-
dc.contributor.daisngid27812973-
dc.contributor.daisngid2696001-
dc.contributor.daisngid6183033-
dc.contributor.daisngid14738520-
dc.description.numberofpages8en_US
dc.utils.revisionSíen_US
dc.contributor.wosstandardWOS:Gonzalez, DA-
dc.contributor.wosstandardWOS:Diaz, BB-
dc.contributor.wosstandardWOS:Perez, MDR-
dc.contributor.wosstandardWOS:Hernandez, AG-
dc.contributor.wosstandardWOS:Chico, BND-
dc.contributor.wosstandardWOS:de Leon, AC-
dc.date.coverdateSeptiembre 2010en_US
dc.identifier.ulpgcSíen_US
dc.description.jcr2,511
dc.description.jcrqQ3
dc.description.scieSCIE
item.fulltextSin texto completo-
item.grantfulltextnone-
Appears in Collections:Reseña
Show simple item record

SCOPUSTM   
Citations

131
checked on Jun 8, 2025

WEB OF SCIENCETM
Citations

115
checked on Feb 8, 2026

Page view(s)

57
checked on Jan 10, 2026

Google ScholarTM

Check

Altmetric


Share


Export metadata


Items in accedaCRIS are protected by copyright, with all rights reserved, unless otherwise indicated.