Please use this identifier to cite or link to this item: http://hdl.handle.net/10553/51948
DC FieldValueLanguage
dc.contributor.authorCabrera, J.en_US
dc.contributor.authorFalcón, L.en_US
dc.contributor.authorGorriz, E.en_US
dc.contributor.authorPardo, M. D.en_US
dc.contributor.authorGranados, R.en_US
dc.contributor.authorQuinones, A.en_US
dc.contributor.authorMaynar Moliner, Manuelen_US
dc.date.accessioned2018-11-25T14:53:58Z-
dc.date.available2018-11-25T14:53:58Z-
dc.date.issued2001en_US
dc.identifier.issn0017-5749en_US
dc.identifier.urihttp://hdl.handle.net/10553/51948-
dc.description.abstractBackground-Some cirrhotic patients with tense ascites who undergo paracentesis develop a circulatory dysfunction syndrome, manifested by an increase in plasma renin activity. Recently, a significant inverse correlation between postparacentesis changes in plasma renin activity and systemic vascular resistance has been demonstrated in these patients, suggesting that peripheral arterial vasodilatation could be responsible for this circulatory dysfunction, but the mechanisms by which tense ascites removal induces such changes are unknownAim-To investigate the role of a decrease in intra-abdominal pressure (IAP) in the development of early postparacentesis haemodynamic changes Methods-Eleven cirrhotic patients with tense ascites received a large volume paracentesis. A specially designed pneumatic girdle was used to compress the abdomen to avoid a decrease in IAP during ascites removal. Haemodynamic studies were performed before paracentesis, one hour after ascites flow stopped, and 30 minutes after pneumatic girdle deflationResults-When LAP was maintained at its original level, no haemodynamic changes were observed, despite large volume paracentesis. However, a significant decrease in systemic vascular resistance was seen immediately after pneumatic girdle deflationConclusions-Early haemodynamic changes after paracentesis are avoided if IAP is maintained at its original level. The abrupt decrease in IAP could be the trigger for the development of the initial haemodynamic changes that eventually produce postparacentesis circulatory dysfunction.en_US
dc.languageengen_US
dc.relation.ispartofGuten_US
dc.sourceGut[ISSN 0017-5749],v. 48, p. 384-389 (Abril 2001)en_US
dc.subject32 Ciencias médicasen_US
dc.subject3205 Medicina internaen_US
dc.subject.otherLarge-Volume Paracentesisen_US
dc.subject.otherTherapeutic Paracentesisen_US
dc.subject.otherHepatic Hemodynamicsen_US
dc.subject.otherPlasma Expandersen_US
dc.subject.otherAlbuminen_US
dc.subject.otherDextran-70en_US
dc.subject.otherSingleen_US
dc.subject.otherTrialen_US
dc.titleAbdominal decompression plays a major role in early postparacentesis haemodynamic changes in cirrhotic patients with tense ascitesen_US
dc.typeinfo:eu-repo/semantics/Articleen_US
dc.typeArticleen_US
dc.relation.conference48th Annual Meeting of the American-Association-for-the-Study-of-Liver-Diseases-
dc.identifier.doi10.1136/gut.48.3.384en_US
dc.identifier.scopus0035056547-
dc.identifier.isi000167239500022-
dc.contributor.authorscopusid7202882445-
dc.contributor.authorscopusid10439154400-
dc.contributor.authorscopusid7004266901-
dc.contributor.authorscopusid7103359396-
dc.contributor.authorscopusid57196983681-
dc.contributor.authorscopusid38061922100-
dc.contributor.authorscopusid7005962555-
dc.description.lastpage389en_US
dc.description.firstpage384en_US
dc.relation.volume48en_US
dc.investigacionCiencias de la Saluden_US
dc.type2Artículoen_US
dc.contributor.daisngid30404347-
dc.contributor.daisngid32206256-
dc.contributor.daisngid1447218-
dc.contributor.daisngid3455912-
dc.contributor.daisngid23424076-
dc.contributor.daisngid29560840-
dc.contributor.daisngid202780-
dc.description.numberofpages5en_US
dc.utils.revisionen_US
dc.contributor.wosstandardWOS:Cabrera, J-
dc.contributor.wosstandardWOS:Falcon, L-
dc.contributor.wosstandardWOS:Gorriz, E-
dc.contributor.wosstandardWOS:Pardo, MD-
dc.contributor.wosstandardWOS:Granados, R-
dc.contributor.wosstandardWOS:Quinones, A-
dc.contributor.wosstandardWOS:Maynar, M-
dc.date.coverdateAbril 2001en_US
dc.identifier.conferenceidevents120290-
dc.identifier.ulpgcen_US
dc.contributor.buulpgcBU-MEDen_US
dc.description.jcr6,17-
dc.description.jcrqQ1-
dc.description.scieSCIE-
item.grantfulltextnone-
item.fulltextSin texto completo-
crisitem.event.eventsstartdate07-11-1997-
crisitem.event.eventsenddate12-11-1997-
crisitem.author.deptGIR Tecnología Médica y Audiovisual-
crisitem.author.deptIU de Investigaciones Biomédicas y Sanitarias-
crisitem.author.deptDepartamento de Ciencias Médicas y Quirúrgicas-
crisitem.author.orcid0000-0001-9154-0712-
crisitem.author.parentorgIU de Investigaciones Biomédicas y Sanitarias-
crisitem.author.fullNameMaynar Moliner, Manuel-
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