Identificador persistente para citar o vincular este elemento: http://hdl.handle.net/10553/51247
Campo DC Valoridioma
dc.contributor.authorBatista, M.
dc.contributor.authorReyes, R.
dc.contributor.authorSantana, M.
dc.contributor.authorAlamo, D.
dc.contributor.authorVilar, J.
dc.contributor.authorGonzález, F.
dc.contributor.authorCabrera, F.
dc.contributor.authorGracia, A.
dc.date.accessioned2018-11-24T22:49:24Z-
dc.date.available2018-11-24T22:49:24Z-
dc.date.issued2011
dc.identifier.issn0936-6768
dc.identifier.urihttp://hdl.handle.net/10553/51247-
dc.description.abstractThis study assessed the efficacy of aglepristone at inducing parturition in pregnant goats. Six experimental groups were defined: group A-5 (n = 12), group A-3.3 (n = 12), group A-2.5 (n = 12) and group A-1.5 (n = 12) in which goats were injected SC once with 5.0, 3.3, 2.5 and 1.5 mg of aglepristone per kg body weight of goat, respectively, group L (n = 11), which was treated IM with 3.75 mg of luprostiol; and group Ct (n = 11), which was injected SC with 1 ml of saline solution. Different parameters associated with parturition were thereafter investigated. In addition, plasma progesterone concentrations were defined after treatments till parturition. Aglepristone effectively induced parturition in all of the goats. In the A-5, A-3.3 and A-2.5 groups, the time to parturition was around 30-34 h, and the majority of goats (97.2%, 35/36) started kidding between 25 and 40 h after the aglepristone injection. However, the goats in group A-1.5 showed a significantly (p < 0.01) higher time to parturition (mean: 46.8 h). Overall, the incidence of dystocia registered in aglepristone-induced goats (20.8%, 10/48) and luprostiol-induced goats was not different from that observed after a spontaneous parturition. The percentage of live kids was very similar between A-5, A-3.3, A. 2.5 and L groups (95.7, 95.3, 95.0 and 96.3%, respectively) but was higher that observed in the control (83.4%) and A-1.5 (81.2%) groups. In addition, no maternal mortality was registered in any groups. No changes in plasma progesterone were observed during the first 24 h after treatment, and high plasma progesterone concentrations were present at kidding (6.7, 5.5, 4.5 and 3.6 ng/ml for groups A-5, A-3.3, A-2.5 and A-1.5, respectively), confirming that aglepristone does not induce parturition via luteolysis. This study demonstrates that aglepristone can be used to induce parturition in goats with satisfactory efficacy, inducing pregnancy termination without direct or immediate modifications of luteal function.
dc.publisher0936-6768
dc.relation.ispartofReproduction in Domestic Animals
dc.sourceReproduction in Domestic Animals[ISSN 0936-6768],v. 46, p. 882-888
dc.subject.otherPregnancy Termination
dc.subject.otherProstaglandin-F2-Alpha
dc.subject.otherProgesterone
dc.subject.otherMifepristone
dc.subject.otherActivation
dc.subject.otherLuteolysis
dc.subject.otherAbortion
dc.subject.otherRelaxin
dc.subject.otherBitches
dc.titleInduction of Parturition with Aglepristone in the Majorera Goat
dc.typeinfo:eu-repo/semantics/Articlees
dc.typeArticlees
dc.identifier.doi10.1111/j.1439-0531.2011.01759.x
dc.identifier.scopus80052638245
dc.identifier.isi000295087300020
dc.contributor.authorscopusid6506315360
dc.contributor.authorscopusid37023320000
dc.contributor.authorscopusid35936129800
dc.contributor.authorscopusid6507009446
dc.contributor.authorscopusid7005533720
dc.contributor.authorscopusid57194243767
dc.contributor.authorscopusid7006639324
dc.contributor.authorscopusid7006185082
dc.description.lastpage888
dc.description.firstpage882
dc.relation.volume46
dc.type2Artículoes
dc.contributor.daisngid30341669
dc.contributor.daisngid5586927
dc.contributor.daisngid2746984
dc.contributor.daisngid1791005
dc.contributor.daisngid2813993
dc.contributor.daisngid30452093
dc.contributor.daisngid5356612
dc.contributor.daisngid2123231
dc.contributor.wosstandardWOS:Batista, M
dc.contributor.wosstandardWOS:Reyes, R
dc.contributor.wosstandardWOS:Santana, M
dc.contributor.wosstandardWOS:Alamo, D
dc.contributor.wosstandardWOS:Vilar, J
dc.contributor.wosstandardWOS:Gonzalez, F
dc.contributor.wosstandardWOS:Cabrera, F
dc.contributor.wosstandardWOS:Gracia, A
dc.date.coverdateOctubre 2011
dc.identifier.ulpgces
dc.description.sjr0,776
dc.description.jcr1,356
dc.description.sjrqQ2
dc.description.jcrqQ2
dc.description.scieSCIE
item.grantfulltextnone-
item.fulltextSin texto completo-
crisitem.author.deptGIR IUIBS: Medicina Veterinaria e Investigación Terapéutica-
crisitem.author.deptIU de Investigaciones Biomédicas y Sanitarias-
crisitem.author.deptDepartamento de Patología Animal, Producción Animal, Bromatología y Tecnología de Los Alimentos-
crisitem.author.deptGIR IUIBS: Medicina Veterinaria e Investigación Terapéutica-
crisitem.author.deptIU de Investigaciones Biomédicas y Sanitarias-
crisitem.author.deptDepartamento de Patología Animal, Producción Animal, Bromatología y Tecnología de Los Alimentos-
crisitem.author.deptGIR IUSA-ONEHEALTH 5: Reproducción Animal, Oncología y Anestesiología Comparadas-
crisitem.author.deptIU de Sanidad Animal y Seguridad Alimentaria-
crisitem.author.deptDepartamento de Patología Animal, Producción Animal, Bromatología y Tecnología de Los Alimentos-
crisitem.author.deptGIR IUSA-ONEHEALTH 5: Reproducción Animal, Oncología y Anestesiología Comparadas-
crisitem.author.deptIU de Sanidad Animal y Seguridad Alimentaria-
crisitem.author.deptDepartamento de Patología Animal, Producción Animal, Bromatología y Tecnología de Los Alimentos-
crisitem.author.orcid0000-0001-9753-4786-
crisitem.author.orcid0000-0002-2060-2274-
crisitem.author.parentorgIU de Investigaciones Biomédicas y Sanitarias-
crisitem.author.parentorgIU de Investigaciones Biomédicas y Sanitarias-
crisitem.author.parentorgIU de Sanidad Animal y Seguridad Alimentaria-
crisitem.author.parentorgIU de Sanidad Animal y Seguridad Alimentaria-
crisitem.author.fullNameBatista Arteaga, Miguel-
crisitem.author.fullNameVilar Guereño, José Manuel-
crisitem.author.fullNameCabrera Martín, Fernando-
crisitem.author.fullNameGracia Molina, Anselmo-
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