|Title:||Bone disease in alcohol abuse||Authors:||Sosa Henriquez, M.
Betancor León, P.
Font The Mora Turon, A.
Navarro Rodríguez, María del Carmen
|UNESCO Clasification:||611301 Alcoholismo
3212 Salud pública
|Issue Date:||1986||Journal:||Annals of internal medicine||Abstract:||To the editor: Bikle and associates (1) have recently emphasized the relation between bone disease and alcoholism. In their eight patients, they found a marked reduction in active bone resorption and bone formation. Nevertheless, serum levels of calcium, phosphorus, magnesium, calcifediol (25-hydroxyvitamin D3), calcitriol (1α, 25-dihydroxyvitamin D3), parathyroid hormone, and nephrogenous cyclic adenosine monophosphate; and urinary levels of calcium, phosphorus, and magnesium; and maximum tubular resorption of phosphate were normal. The authors therefore suggested that the bone disease in these patients was due to an inhibition of bone remodeling through a mechanism independent of the calciotropic hormones.||URI:||http://hdl.handle.net/10553/50809||ISSN:||0003-4819||DOI:||10.7326/0003-4819-104-6-893_1||Source:||Annals of internal medicine [ISSN 0003-4819], v. 104 (6), p. 893, (Junio 1986)|
|Appears in Collections:||Comentario|
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