Identificador persistente para citar o vincular este elemento: http://hdl.handle.net/10553/44965
Título: Depletion of serum alternative complement pathway activity in gilthead seabream caused by α-tocopherol and n-3 HUFA dietary deficiencies
Autores/as: Montero, D. 
Tort, L.
Izquierdo, M. S. 
Robaina, L. 
Vergara, J. M. 
Clasificación UNESCO: 251092 Acuicultura marina
Palabras clave: Alternative complement
Stress
Sparus aurata
Cortisol
Vitamin E, et al.
Fecha de publicación: 1998
Editor/a: 0920-1742
Publicación seriada: Fish Physiology and Biochemistry 
Resumen: Studies on the alternative complement pathway (ACP) activity in fish have suggested the particular relevance of this pathway as a non-specific immune mechanism being its activation directly related with the availability of certain nutrients. In the present study ACP activity in gilthead seabream fed different dietary levels of a-tocopherol and n-3 HUFA deficiencies was studied under different stress conditions (overcrowding and repetitive chasing). A reduction in ACP activity was found when diets with deficiencies in α-tocopherol and n-3 HUFA were assayed at any of the experimental conditions assayed. Levels of plasma cortisol were similar to those of fish after crowding stress subjected to dietary deficiencies at low stocking density, suggesting that dietary deficiencies produced a chronic elevation of cortisol. When fish were subjected to repetitive chasing, plasma cortisol from fish fed control diet showed a progressive increase whereas plasma cortisol levels from both dietary deficient groups showed a transitory peak 4 days after the beginning of the repetitive chasing. However, ACP activity levels of fish fed deficient diets remained lower than the control group and constant during chasing period. These results suggest that both a-tocopherol and n-3 HUFA nutritional deficiencies rather than stress are an important determinant of fish complement activity.
URI: http://hdl.handle.net/10553/44965
ISSN: 0920-1742
DOI: 10.1023/A:1007734720630
Fuente: Fish Physiology and Biochemistry [ISSN 0920-1742], v. 18 (4), p. 399-407
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